11. What would happen if histone H1 could be selectively (30 nm) extracted from compacted chrinin?
A) 30 nm fibers were expanded to produce thicker and longer beaded filaments.
To produce a thicker and more extended filament form, b) use 30 nm fibers.
c) Fibers with a diameter of 30 nm are completely destroyed except for their nucleotides.
d. Fibers with a diameter of 30 nm break down into small pieces.
e) 30 nm fibers disintegrate into large pieces.
12) Is there experimental evidence that the N-terminal tails of histones are involved in histone formationa Higher order chromatin structure plays a role
A) Histones are denatured by tails
b. Acetylated tails lead to resolution (30 nm) fibers
c. Chromatin fibers from H4 histones without ends cannot fold into higher order fibers
d) Chromatin fibers from H4 histones with longer ends cannot fold into higher order fibers.
e) Chromatin fibers without ends have longer tails.
13) DNA loops for 80 – 100 nm fibers begin and end with _______.
a) AT-rich sequences
b) GC-rich sequences (* )c) GT-rich sequences
d. Poly(A), chains
e) Single chain part ( *)14) Which gene is responsible for the inactivation of X chromosome RNA?
d. Internal Revenue Service
15) What is the difference between regulatory and noncoding RNAs that are thought to cause X chromosome activation?
It is much larger than other non-coding RNAs.
b. It is smaller than non-coding RNA.
It is a tighter coil than non-coding RNA.
It has more uracil than non-coding RNA.
e) This RNA has more uracil than the adenine of the non-coding RNA.
16) Some genes of the X chromosome survive inactivation through unknown mechanisms. What genes does this group consist of? Why are they still activated?
(a) Genes deleted on the X chromosome are still expressed in equal amounts in both sexes
b) Genes also present on the Y chromosome. They express e (centromere) equally in both sexes; they express equally in males and females
17) Evidence suggests that while the XIST genes may be necessary to inactivate the X chromosome, it may not be necessary to turn them off to keep a generation alive. ( * )One. Tumor cells do not maintain the inactivation of the X chromosome from generation to generation.
b) An inactive X chromosome can be found in tumor cells from women whose XIST gene has been deleted.
C) Certain tumor cells found in females are inactivated by the X chromosome in which the XIST gene is duplicated.
d. Some people are color blind and some are not.
e. Some cancer cells found in women do not have an X chromosome.
18) What causes X chromosome activation?
a) Repressive histone modification
b) RNA inactivation
c) DNA methylation (* ) d) Local DNA denaturation
e) DNA methylation and repressive histone modification
19) What evidence suggests that RNAi plays a role in heterochromatinization?
a) Loss of RNAi components affects histone H3K9 methylation and heterochromatinylation.
b) Addition of RNAi components affects histone H3K9 methylation and heterochromatinylation.
c) Deletion of the RNAi component enhances histone H3K9 methylation and heterochromatinization.
d) Mutations in RNAi components denature and heterochromize histone H3K9 methylation.
20. What is the advantage of highly condensed DNA from mitotic chromosomes
a) This state enables gene expression during mitosis.
b) The highly concentrated form facilitates delivery of the complete DNA package to each daughter cell.
c. The condensed form allows chromosomes to fuse more easily.
d. When a cell enters mitosis, it can replicate more quickly in a condensed state.
e) In the condensed state, pre-mitotic DNA repair is possible.
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